Patients who manifest global cognitive impairment before treatment and patients who experience prolonged disorientation in the acute postictal period may be the most vulnerable to persistent retrograde amnesia for autobiographical information. In general, these relationships were maintained regardless of technical parameters in the administration of the ECT. Pre-ECT global cognitive status and the duration of postictal disorientation were strong predictors of the magnitude of retrograde amnesia in the week after the course of ECT and at 2-month follow-up. Comparisons between forms of amnesia: some deficits are unique. Implicit memory is mostly associated with the habits you acquire throughout your life like texting. We conducted a prospective, naturalistic. Firstly, memory can be divided into two broad classifications which are implicit (procedural) memory and explicit (declarative) memory. Despite ongoing controversy, there has never been a large-scale, prospective study of the cognitive effects of electroconvulsive therapy (ECT). Retrograde amnesia was assessed at these same time points with the Autobiographical Memory Interview. Two forms of human amnesia: an analysis of forgetting. Amnesia is when brain damage, disease or psychological trauma causes memory deficit. Global cognitive status was assessed with the modified Mini-Mental State examination before treatment, during the week after termination of treatment, and 2 months after treatment ended. Orientation recovery time was assessed at virtually every session during the course of ECT. The type and amount of ECT appear to be factors in memory loss, so we try to be as conservative as possible when treating. Seventy-one inpatients with major depressive disorder were randomly assigned to four ECT conditions that varied in electrode placement (right unilateral versus bilateral) and stimulus dosage (low versus high intensity). Rarely, some patients describe much more significant retrograde memory loss, but with advances in ECT, this is much less common and usually only occurs in cases of severe illness and long or repeated courses of ECT. The authors examined patients' pretreatment global cognitive status and postictal orientation recovery time as potential predictors of the magnitude of retrograde amnesia for autobiographical memories after ECT. The memory can be either wholly or partially lost due to the extent of damage that was caused. However, there is limited information regarding individual differences in vulnerability to these side effects. Amnesia is a deficit in memory caused by brain damage or brain diseases, but it can also be temporarily caused by the use of various sedatives and hypnotic drugs. Substantial progress has been made in identifying how the treatment parameters used in ECT impact on cognitive side effects.
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